Introduction Procedure Results Figures Discussion References	Results As expected, the control group continued to develop normally for the duration of the experiment (Fig. 1 & 2). The only malformations in this group were attributed to mechanical harm suffered during the de-jellying process, but this information will be discounted as insignificant as the damaged embryos continued to develop more or less normally. The embryos exposed to both low (30 uL) and high (60uL) levels of ethanol were unable to survive longer than 48 hours. Prior to the fatal disassociations suffered by the embryos of these two experimental groups (Fig. 3), development seemed to appear normal. The 50 uM cyclopamine experimental group also suffered 100% fatalities after 48 hours of exposure to the teratogen. This was, in part, due to mechanical trauma suffered during de-jellying. However, the undamaged embryos also exploded somewhere between H&H stages 33 and 37, most likely due to mechanical harm suffered during the de-jellying process. Prior to embryo death, slight malformations in the tail bud and body trunk region were detected in this experimental group (Fig. 4). The most conclusive experimental evidence from this experiment can be observed in the 100 uM experimental group. Survival rate was 100% in this group, and development was tracked for a full 72 hours after exposure to the teratogens. Noted abnormalities include slight cranial malformations, including a smaller head and underdeveloped gill structures; a much shorter body trunk; and severe tail truncation (Fig. 5 & 6).